Search results (17)
« Back to PublicationsAberrant dynein function promotes TDP-43 aggregation and upregulation of p62 in male mice harboring transgenic human TDP-43.
Journal article
Christoforidou E. et al, (2023), Amyotrophic lateral sclerosis & frontotemporal degeneration, 1 - 10
An ALS-linked mutation in TDP-43 disrupts normal protein interactions in the motor neuron response to oxidative stress
Journal article
Feneberg E. et al, (2020), Neurobiology of Disease, 144, 105050 - 105050
Mice Carrying ALS Mutant TDP-43, but Not Mutant FUS, Display In Vivo Defects in Axonal Transport of Signaling Endosomes.
Journal article
Sleigh JN. et al, (2020), Cell reports, 30, 3655 - 3662.e2
Neuronal over-expression of Oxr1 is protective against ALS-associated mutant TDP-43 mislocalisation in motor neurons and neuromuscular defects in vivo
Journal article
Williamson MG. et al, (2019), Human Molecular Genetics, 28, 3584 - 3599
Single-copy expression of an amyotrophic lateral sclerosis-linked TDP-43 mutation (M337V) in BAC transgenic mice leads to altered stress granule dynamics and progressive motor dysfunction
Journal article
Gordon D. et al, (2019), Neurobiology of Disease, 121, 148 - 162
The interactome of human TDP-43 in a cellular model of amyotrophic lateral sclerosis
Conference paper
Feneberg E. et al, (2019), EUROPEAN JOURNAL OF NEUROLOGY, 26, 221 - 222
TDP-43 is cytoplasmically mislocalized and associated with impaired stress responses and survival of primary neurons from symptomatic amyotrophic lateral sclerosis (ALS) mice
Conference paper
Farrimond L. et al, (2016), EUROPEAN JOURNAL OF NEUROLOGY, 23, 390 - 391
Absence of wide-spread mis-splicing in the preclinical phase of a native promoter driven TDP-43 mouse model of ALS
Conference paper
Scaber J. et al, (2016), EUROPEAN JOURNAL OF NEUROLOGY, 23, 797 - 797
Absence of wide-spread mis-splicing in the preclinical phase of a native promoter driven TDP-43 mouse model of ALS
Conference paper
Scaber J. et al, (2016), NEUROMUSCULAR DISORDERS, 26, S31 - S31
TARDBP pathogenic mutations increase cytoplasmic translocation of TDP-43 and cause reduction of endoplasmic reticulum Ca2+ signaling in motor neurons
Journal article
Mutihac R. et al, (2015), Neurobiology of Disease, 75, 64 - 77
Fusion between human mesenchymal stem cells and rodent cerebellar Purkinje cells
Journal article
Kemp K. et al, (2011), Neuropathology and Applied Neurobiology, 37, 166 - 178
Human Mesenchymal Stem Cells Infiltrate the Spinal Cord, Reduce Demyelination, and Localize to White Matter Lesions in Experimental Autoimmune Encephalomyelitis
Journal article
Gordon D. et al, (2010), Journal of Neuropathology & Experimental Neurology, 69, 1087 - 1095
Human mesenchymal stem cells abrogate experimental allergic encephalomyelitis after intraperitoneal injection, and with sparse CNS infiltration
Journal article
Gordon D. et al, (2008), Neuroscience Letters, 448, 71 - 73
Antisense suppression of tau in cultured rat oligodendrocytes inhibits process formation
Journal article
Gordon D. et al, (2008), Journal of Neuroscience Research, 86, 2591 - 2601